By American Academy of Ophthalmology, Ramana S. Moorthy MD
Starts with an summary of immune-mediated eye sickness, summarizing uncomplicated immunologic innovations, ocular immune responses and specified subject matters in ocular immunology. Discusses the scientific method of uveitis and reports noninfectious (autoimmune) and infectious kinds of uveitis, with an improved part on viral uveitis and new fabric on infectious and noninfectious scleritis.
Enhanced detection of infectious brokers by way of immunologic and genetic equipment and biologic therapeutics are designated. additionally covers endophthalmitis, masquerade syndromes, problems of uveitis and ocular facets of AIDS.
Upon crowning glory of part nine, readers can be capable to:
Identify normal and particular pathophysiologic procedures that have an effect on the constitution and serve as of the uvea, lens, intraocular cavities, retina, and different tissues in acute and protracted intraocular inflammation
Differentiate and determine infectious and noninfectious uveitic entities
Choose acceptable exam suggestions and proper ancillary stories in response to no matter if an infectious or noninfectious reason is suspected
Read Online or Download 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis PDF
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Additional resources for 2014-2015 Basic and Clinical Science Course (BCSC): Section 9: Intraocular Inflammation and Uvetis
Because the response is delayed by 24 hours, it is considered DH and, in this case, a specific form of DH called contact hypersensitivity. Primary and secondary response to tuberculosis The primary and secondary immune response arcs can occur at different sites, as with the immunologic mechanisms of the first and second encounter with Mycobacterium tuberculosis antigens. The afferent phase of the primary response occurs after the inhalation of the live organisms, which proliferate slowly within the lung.
The T lymphocytes interact with the macrophageingested bacteria, and cytokines are released that activate neighboring macrophages to fuse into giant cells, forming caseating granulomas. Meanwhile, some of the effector T lymphocytes home to other lymph nodes throughout the body, where they become inactive memory T lymphocytes, trafficking and recirculating throughout the secondary lymphoid tissue. A secondary response using the immune response arc of the skin is the basis of the tuberculin skin test to diagnose tuberculosis.
Processing Phase The conversion of the antigenic stimulus into an immunologic response through priming of naive B and T lymphocytes within the lymph nodes and spleen constitutes the processing phase of the immune response arc. This is also called activation, or sensitization, of 38 • lntraocular Inflammation and Uveitis 1 HLA 2 Infected APC 4 3 Costimulation = ,- - Antigen receptor CD8 T lymphocyte Figure 2-2 Class I-dependent antigen-presenting cells (APCs) . 1, APC is infected by a virus, which causes the cell to synthesize virus-associated peptides that are present in the cytosol.